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Middot; before taking adoxa, tell your doctor if you are taking any of the following drugs: · cholestyramine questran ; or colestipol colestid · an antacid such as tums, rolaids, milk of magnesia, maalox, and others; · a product that contains bismuth subsalicylate such as pepto-bismol; · minerals such as iron, zinc, calcium, magnesium, and over-the-counter vitamin and mineral supplements; · carbamazepine tegretol, carbatrol, epitol · phenytoin dilantin, phenytek · didanosine videx · a blood thinner such as warfarin coumadin · sucralfate carafate · a barbiturate such as phenobarbital, mephobarbital mebaral ; , secobarbital seconal ; , or pentobarbital nembutal · a penicillin antibiotic such as amoxicillin amoxil, trimox, others ; , penicillin beepen-vk, pen-vee k, veetids, others ; , dicloxacillin dynapen ; , carbenicillin geocillin ; , oxacillin bactocill ; , and others; or · methoxyflurane an inhaled anesthetic gas used during surgery. Figure 7. Example showing the first 7 values of a linear ramp table. Enteric Contrast Material in Children Before CT risk [1418]. There is even controversy concerning the terminology, such as conscious sedation, used to describe the degree of sedation [19]. A number of health care professionals consider the degree of pharyngeal depression and the risk of vomiting and aspiration to be less with drugs such as pentobarbital than with a general anesthetic [1418]. Certainly, the incidence of vomiting, degree of pharyngeal depression, and risk of aspiration are different for different sedatives [15]. In this sense, our data apply to the use of only pentobarbital and choral hydrate. Because we have few patients who were sedated with choral hydrate, our data mainly pertain to patients sedated with pentobarbital. Concerning the incidence of vomiting, pharyngeal depression, and risk of aspiration related specifically to pentobarbital administration, we could find no published data except the mention of vomiting as a potential adverse effect in the package insert Nembutal; Abbott Laboratories, North Chicago, IL ; . To our knowledge, no cases have been published of aspiration from enteric contrast material leading to complications in pediatric patients who were sedated. In our review of 5 years of experience concerning the administration of enteric contrast material before sedation in a busy pediatric body imaging service, no cases were discovered in which aspiration was noted or led to clinically significant adverse effects. Our study is limited by a number of factors. The study is retrospective in nature, and the detection of aspiration was dependent on the policy that all adverse events be recorded in the medical record and incident reports be filed. Although none of the pediatric radiologists in our body imaging section or the radiology sedation nurses recall a case of aspiration related to enteric contrast material, and we think that it is unlikely that a significant incident of aspiration could occur without documentation, the inclusion of such an event is dependent on the reporting policies of the department. Second, a control group was not included in our study. In future studies, a control group composed of children who were sedated for IV contrast-enhanced CT studies but did not require oral contrast material CT of the chest, neck, or head ; could be used to determine whether oral contrast material does or does not contribute to a higher degree of aspiration. Third, despite a large number of abdominal CT examinations that were performed at our institution during the 5-year period of the study, only 367 were performed with sedation. This finding shows that most children who undergo CT of the abdomen do not require sedation. A multicenter study may be advantageous in providing a larger number of subjects. Finally, as previously mentioned, the conclusions from this study can be applied strictly to only those drugs used in our sedation program. Future studies may be needed to see whether the same conclusions can be drawn concerning other sedatives. All patients in our study also received IV contrast material, which can lead to nausea and vomiting. CT examinations that require oral but not IV contrast material are uncommon but may have an even more decreased risk of aspiration than scans obtained with IV contrast material. In conclusion, the practice of administering oral contrast material before sedation for abdominal CT in children appears to be safe when using the sedation drugs and protocols in place at our institution. Further study into the safety of this practice should be undertaken.

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SELECTED DRUG DOSAGES * Dissociative Agents and Combinations Ketamine 11-33 mg kg, I.M., I.V. Ketamine 10-33 mg kg hr IV infusion Ketamine 22-33 mg kg: Acetylpromazine 1.1 mg kg I.M. Ketamine 15 mg kg: Diazepam 2 mg kg, I.M. Ketamine 20 mg kg: Xylazine 2 mg kg I.M. Ketamine 11 mg kg: Fentanyl-Droperidol Invovar-Vet ; 1 ml 14 kg I.M. Ketamine 15 mg kg: Azaperone 2 mg kg, I.M. Ketamine 2 mg kg: Xylazine 2 mg kg: Oxymorphone .075 mg kg I.V. 2x dose for I.M. ; Ketamine 20 mg kg: Climazolam .5 - 1.0 mg kg I.M. Tiletamine - Zolazepam Telazol ; - 4-6 mg kg I.M. Tiletamine - Zolazepam Telazol ; - 4-6 mg kg I.M: Xylazine 2.2 mg kg II. Barbiturates Pentobarbital 20-40 mg kg I.V.; 5-40 mg kg hr continuous I.V. infusion Thiopental 6.6 - 30 mg kg I.V.; 3-30 mg kg hr continuous I.V. infusion III. Miscellaneous Injectable Restraint Agents Azaperone 2-8 mg kg I.M. "Chloralose - 55-100mg kg I.V. Etomidate 4-8 mg kg IV Etorphine Acetylpromazine Imobilon ; .245 mg 10 kg: Diprenorphine Revivon ; .3 mg kg Ketamine: Xylazine: Glyceryl guaiacolate 1 ml kg see text for mixture ; Midazolam - 100-500 mcg kg I.M. Metomidate 4 mg kg IV Meperidine 1 mg lb: Azaperone 1 mg lb followed in 20 minutes by Ketamine 10 mg lb: Morphine 1 mg lb Propofol 0.83-1.66 mg kg I.V.; 12-20 mg kg hr continuous IV infusion IV. Analgesic Fentanyl - .02 - .05 mg kg IM q 2h; 30-100 mcg kg hour IV drip Sufentanyl - 5-10 mcg kg IM q 2h; 10-30 mcg kg hour IV drip Buprenorphine - .01-.05 mg kg range .005 - .1 mg kg ; IM I. No. of seizures per night Normal Sparse bilateral frontotemporal sharp waves, intermittent frontotemporal theta activity Not done Not localizing Not done Normal Sparse bifrontal slow sharp waves Sparse bifrontal slow sharp waves.

1. SODI PALLARES D: New bases of electrocardiography. St Louis, MO, C.V. Mosby Co, 1956, p. 404. 2. MEDRANO GA, DE MICHELI A, ARANDA A, ITURRALDE P: Es an vlido el concepto de "salto de onda"? Arch Inst Cardiol Mex 2000; 70 1 ; : 19-29. 3. SODI PALLARES D, BISTENI A, MEDRANO GA: Electrocardiografa y vectocardiografa deductivas. Mxico. La Prensa Mdica Mexicana, 1964; T.I: 111-116. 4. MEDRANO GA, BISTENI A, BRANCATO RW, PILEGGI F, SODI PALLARES D: The activation of the interventricular septum in the dog's heart under normal conditions and in bundle branch block. Ann NY Acad Sci 1957; 65: 804-817. CRDENAS M, SANZ G, LINARES JC, ZAMORA C, MEDRANO GA, ESTANDA A: Diagnstico electrocardiogrfico de infarto del miocardio con estimulacin endocrdica del ventrculo derecho por and pentostatin. In looking at the Car of Tomorrow, Foresight Vehicle, and ACE projects See Appendix 1 ; , solutions should be acceptable for buses and trucks, as well as cars. This added application requires greater transferability between transportation vehicles. A more universal approach must be utilized with other programs. The ACE project has given individual assignments to each company. Isuzu will work on engine and capacitor arrangement for trucks, while Nissan has been assigned fuel cell and battery research for the passenger car, and so on through each participating company. [22] Comparatively, PNGV has allowed each company to work on all technologies collaboratively. The Foresight and Car of Tomorrow program are looking for technologies that will be transferable between different transportation modes. The Foresight program, however, specifies that "The technologies will be suitable for all types of road vehicles from small cars to large goods vehicles." [10] Eventually the PNGV technologies may be applied to other transportation vehicles, but presently this is not a main objective. Collaboration Differences.

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Six male dogs weighing 24.81.3 meanSEM ; kg were used in this study. The dogs were anesthetized with pentobarbital sodium 30 mg kg i.v. ; and chronic indwelling Tygon catheters 0.05 in. i.d., 0.09 in. o.d., Norton Plastics, Akron, Ohio ; were placed in the femoral arteries and veins. The tips of the femoral arterial catheters were advanced into the aorta distal to the origin of the renal arteries, and the ends of the femoral vein catheters were positioned in the lower inferior vena cava. A silastic elbow prevented kinking of the catheters in the femoral area. The catheters were tunneled subcutaneously and exteriorized at the intrascapular region of the back. Patency of vascular catheters was maintained by flushing with sterile isotonic saline every 2-3 days and by filling the catheters with heparin 1, 000 units ml ; . The catheters were protected within a canvas jacket. Two weeks after surgery, the dogs were placed in metabolic pens and fitted with an aluminum and canvas backpack housing a Statham arterial blood pressure transducer model P23 ID, Statham Laboratories, Inc., Hato Rey, Puerto Rico ; at heart level. Restraining lines from the sides of the pen were connected to the four corners of the backpack; this permitted the dogs to move freely in the cage but not to turn completely around. The electrical connections to the arterial blood pressure transducer and an intravenous infusion line were brought to the top of the cage through a flexible tube attached to the top of the backpack. Continuous intravenous infusions were made through one of the femoral vein catheters by means of a Sage tubing pump model 375A, Sage Instruments, Cambridge and peppermint!
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MATERIALS AND METHODS rVSV. The rVSV was made from reconstitution of the Indiana strain of VSV from DNA 15 ; coding for a positive-sense strand of RNA, as described in detail elsewhere 7 ; . Briefly, the coding sequence for an enhanced redshifted and mammalian codon-corrected GFP EGFP; Clontech ; was fused to the coding sequence of the VSV G protein and incorporated as an extra gene downstream of the unmodified VSV gene. VSV particles contain about 1, 200 molecules of the G protein on the viral surface 39 ; . A recombinant virus that expresses GFP fused to the VSV G protein was generated from a combination of four plasmids that were cotransfected 29 ; into baby hamster kidney cells BHK-21 ; to generate rVSV. vTF73, a recombinant vaccinia virus, was used to provide T7 RNA polymerase, an enzyme necessary for gene expression. rVSV from plates showing a cytopathic effect and GFP expression was titrated on BHK-21 cell monolayers, individual green plaques were isolated, and virus from them was grown on BHK-21 cells. This virus expresses both wild-type G and G-GFP fusion proteins, which form heterotrimers. The heterotrimers are transported to the cell surface and incorporated into budding virions. Intracerebral and transnasal infection. Adult mice, 35 to 40 days old at the time of inoculation, were used for intracerebral injections n 8 ; and nasal application n 9 ; . After anesthetizing the mouse with Nembutal 60 mg kg of body weight ; , a small hole was made in the top of the skull. A Hamilton microsyringe was used to deliver a small volume 0.1 to 0.5 l ; of sterile buffer containing VSV 108 PFU ml ; . The tip of the injection needle was lowered 3 mm down from the top of the skull, 1.5 mm lateral to right of the midline, and the VSV was administered by slow application of pressure. Four mice received injections aimed at the rostral part of the brain striatal area ; , and four mice received injections aimed more caudally at the hippocampal area. After the mice had been anesthetized with pentobarbital Nembutal; 60 mg kg ; , 15 l of sterile medium containing VSV was applied to each of the external nares, resulting in infection of the nasal cavity. At 2 n days n 5 ; postinoculation p.i. ; , mice were given an overdose of anesthetic Nembutal; 100 mg kg ; and perfused transcardially with physiological saline followed by freshly prepared 4% paraformaldehyde. Serial sections with a thickness of 10 to were cut on a freezing sledge microtome through the entire brain. From the serial sections, 1 of every 20 sections was mounted in a cryoprotectant buffer on glass slides, giving a representative view of the entire brain. After a coverslip was added, sections were saved in a freezer until studied. Primary mouse brain culture. Embryonic brain tissue was used for primary neuronal culture. Cells were plated on glass coverslips that had been pretreated with polylysine to enhance the affinity of cells to the substrate. In some experiments, we used cultures of whole brain, and in others, we used selective cultures of single brain regions, often the hippocampus. More details of the culture methodology can be found elsewhere 41 ; . In some experiments, we used cultures of various organs to compare relative VSV infection and production. Animal use in these experiments was approved by the University Committee on Animal Use. Human primary culture. Tissue from human cerebral cortex was used for primary culture. Tissue was removed solely for medical reasons and for the patient's benefit. Tissue was from brain tumors that included both part of the tumor and tissue at the edge of the tumor. Use of the tissue after surgical removal was approved by the Human Investigations Committee of the University. Live brain slices. After animals were given an overdose of Nembutal, brains were removed and cut into 200- m-thick slices. Slices were maintained at the gas-fluid interface on a Millipore membrane at 37oC with 5% CO2. Brain slices were fed with Neurobasal Gibco BRL ; . Slices were infected with rVSV by placing a drop of virus-containing medium on the slice 106 PFU per slice and pergolide.

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Adams, I.B., and Martin, B.R. Cannabis: Pharmacology and toxicology in animals and humans. Addiction 91 11 ; : 1585-1614, November 1996. Agurell, S., Dewey, W.L., and Willett, R.E., eds. The Cannabinoids: Chemical, Pharmacologic, and Therapeutic Aspects. New York: Academic Press, 1984. Agurell, S.; Halldin, M.; Lindgren, J.E.; Ohlsson, A.; Widman, M.; Gillespie, H.; and Hollister, L. Pharmacokinetics and metabolism of delta 1-tetrahydrocannabinol and other cannabinoids with emphasis on man. Pharmacol Rev 38 1 ; : 21-43, March 1986. Barnett, G.; Licko, V.; and Thompson, T. Behavioral pharmacokinetics of marijuana. Psychopharmacology 85 1 ; : 51-56, 1985. Benowitz, N.L., and Jones, R.T. Effect of delta-9-tetrahydrocannabinol on drug distribution and metabolism: Antipyrine, pentobarbital and ethanol. Clin Pharmacol Ther 22 3 ; : 259-268, 1977. Benowitz, N.L., and Jones, R.T. Cardiovascular and metabolic considerations in prolonged cannabinoid administration in man. J Clin Pharmacol 21: 214S-223S, 1981. Benowitz, N.L.; Nguyen, T.; Jones, R.T.; Herning, R.I.; and Bachman, J. Metabolic and psychophysiologic studies of cannabidiol-hexobarbital interaction. Clin Pharmacol Ther 28: 115-120, 1980. Duffy, A., and Milin, R. Case study: Withdrawal syndrome in adolescent chronic cannabis users. J Acad Child Adolesc Psychiatry 35 12 ; : 1618-1621, December 1996. Fehr, K., and Kalant, H., eds. ARF WHO Scientific Meeting on Adverse Health and Behavioral Consequences of Cannabis Use 1981: Toronto, Canada ; Cannabis and Health Hazards: Proceedings of an ARF WHO Scientific Meeting on Adverse Health and Behavioral Consequences of Cannabis Use. Toronto, Canada: Addiction Research Foundation, 1983. Friedman, H.; Klein, T.W.; Newton, C.; and Daaka, Y. Marijuana, receptors and immunomodulation. Adv Exp Med Biol 373: 103-113, 1995. Graham, J.D.P., ed. Cannabis and Health. New York: Academic Press, 1976. Grinspoon, L., and Bakalar, J.B. Marihuana, the Forbidden Medicine. New Haven: Yale University Press, 1993. Gruber, A.J., and Pope, H.G. Cannabis psychotic disorder: Does it exist? J Addict v3 n1 ; : 72-83, Winter 1994. Harvey, D.J., ed. Satellite Symposium on Cannabis 3rd: 1984: Oxford, England ; Marihuana '84: Proceedings of the Oxford Symposium on Cannabis. Washington, DC: IRL Press, 1985. Heishman, S.J.; Stitzer, M.L.; and Yingling, J.E. Effects of tetrahydrocannabinol content on marijuana smoking behavior, subjective reports, and performance. Pharmacol Biochem Behav 34 1 ; : 173-179, September 1989. Herkenham, M. Cannabinoid receptor localization in brain: Relationship to motor and reward systems. In: Kalivas, P.W., and Samson, H.H., eds. The neurobiology of drug and alcohol addiction. Ann N Y Acad Sci 654: 19-32, 1992. Herning, R.I.; Hooker, W.D.; and Jones, R.T. Tetrahydrocannabinol content and differences in marijuana smoking behavior. Psychopharmacology 90 2 ; : 160-162, 1986. Hollister, L.E. Health aspects of cannabis. Pharmacol Rev 38 1 ; : 1-20, March 1986a. Hollister, L.E. Interactions of cannabis with other drugs in man. In: Braude, M.C., and Ginzburg, H.M., eds. Strategies for Research on the Interactions of Drugs of Abuse. National Institute on Drug Abuse Research Monograph 68. DHHS Pub. No. ADM ; 86-1453. Washington, DC: Supt. of Docs., U.S. Govt. Print. Off., 1986b. pp. 110-116. 17.

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Tritide reagents for the synthesis of tritiated natural C compounds 064 Andres Switzerland Expedient incorporation of Tritium into drug candidate 065 Berthelette Canada Use of Tritium-labeled drugs in clinical studies at Abbott Laboratories 066 Rotert U.S.A. Tritium labelling of novel endomorphin analogues containing unnatural alpha- and beta-amino acids 067 Gza Hungary Overview of posters 182, 183, 184 W.J. Wheeler and permax.

Lena Marions, friend and colleague, always there with her valuable support. Christian Fiala, co-author, for all his help and friendship. My co-authors Ann-Kristin Ulfgren, Anneli Stavreus-Evers, Olof Stephansson, Fredrik Granath, Bernhard Watzer, HW Schweer who have been a pleasure to work with. A special thank to Berit Stby for all their help and support. Professor Sten Cekan for statistical analysis and kind help and support. Margareta Hellborg and Lena Elffors Sderlund, research nurses at WHO Centre for their excellent work and highly professional care of the patients and for always giving me kind support and for all the good time we shared. Monika Johansson and Eneli Salomonsson, staff at SESAM and Maijken Ullmark, Marietta Molnar, Eva-Lisa Nordlund, Marianne Racke and Inga-Maj Andersson, staff at C21 for their warm and professional care of patients and kind support. Helena von Hertzen and Paul Van Look at the WHO Centre in Geneva for their kind collaboration. The team at WHO centre, for all your valuable help and kindness. Astrid Hggblad for all her help during my years as a PhD student. Catharina Karlsson for help and friendship . Eva Eneroth I will miss you ; and other colleagues in labour ward for all the nice time we shared. Birgitta Bystrm and other colleagues at the FoU-laboratory for their valuable support during the years. Professor PC Ho, Hong Kong for sharing his ideas about sublingual administration of misoprostol. 48.
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Background: Neurological soft signs NSS ; have become an important research instrument for the assessment of subtle neurological impairments in schizophrenia. However, there are very few studies in first-episode schizophrenia investigating the association of NSS with other relevant markers of the disease, such as cognitive deficits. Methods: 59 neurolepticnaive first-episode schizophrenia patients and 51 healthy individuals, matched for age, gender, and parental socioeconomic status, were included. All subjects were assessed by use and phenazopyridine.
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Fifty-three inongrel dogs of either sex weighing 165 mean + SD ; kg were anesthetized with pentobarbital 25 mg kg i.v. ; , intubated, and connected to.

Kraus J., Gamulescu M.-A., Framme C., Gabel V.-P. Regensburg D ; Photodynamische Therapie bei pathologischer Myopie klinische Ergebnisse Photodynamic therapy in pathologic myopia clinical results Krohne T. U., Kmmerer E., Kopitz J., Holz F. G. Bonn D, Heidelberg D ; Lysosomal proteolytic activities in retinal pigment epithelial cells are reduced by lipid peroxidation products via two different kinds of action Loncarek K., Filipovic T., Stalekar H., Vatavuk Z., Petricek I., Brajac I. Rijeka HR, Zagreb HR ; Sub-tenon application of triamcinolone-acetonide in neovascular age-related macular degeneration Marchanka L. N., Birich T., Fedulov A. S., Krivonosov V. V., Petrov P. T., Trukhatcheva T. V., Savitch A. V., Shkadarevich A. P., Kurganovich A. M., Kharitonchik T. L., Dulub L., Fedulova A. A. Minsk BY ; Photolon in photodynamic therapy of choroidal neovascularization in age-related macular degeneration and myopic maculopathy Morimoto M., Sato T., Watanabe G., Kishi S. Maebashi J ; Laser photocoagulation for juxtafoveal choroidal neovascularization in angioid streaks Morkhat V., Marchanka L. N., Morkhat M. Vitebsk BY, Minsk BY ; New possibilities of diagnosis of age-related macular degeneration Pehlivanl Z., zdek S., Grelik G., Hasanreisoglu B. B. Ankara TR ; Transpupillary Thermotherapy TTT ; for occult choroidal Neovascularization in age-related macular degeneration: 17Months follow-up and phenelzine. Guidelines established by the European Union 86 609 ; . After anesthesia of the rat with pentobarbital sodium 50 mg kg ip ; , the pancreas was dissected and perfused in situ according to the procedure of Leclercq-Meyer et al. 14 ; as adapted in our laboratory 28 ; . Effluent samples were collected from the portal vein, without recycling, at 1-min intervals flow rate, 2 ml min ; and frozen at 20C until the time of assay. The perfusion medium consisted of a Krebs-Henseleit buffer [in mM: 115 NaCl, 4.7 KCl, 2.6 CaCl2, 1.19 H2KPO4, 1.19 MgSO4 7H2O, and 24.9 HNaCO3 gas phase 95% O2-5% CO2; pH 7.4 ; ] supplemented with 4% wt vol ; dextran T-70 Pharmacia LKB Biotechnology, Uppsala, Sweden ; , 0.5% wt vol ; Cohn fraction V bovine albumin Sigma Chemical, St. Louis, MO ; , and glucose 3.2, 5.5, or 11 mM; Sigma ; . Rat amylin Amylin Pharmaceuticals, San Diego, CA ; was dissolved in 0.9% NaCl containing 0.1% bovine albumin Cohn fraction V ; . This solution 17.8 nM ; was prepared daily, immediately before experiments, and when added to the perfusate, the final concentration was 1 nM amylin. After a 35-min equilibration period, baseline samples were collected for 5 min, and, at time 0, normal saline with or without amylin was infused through a sidearm cannula. Glucagon secretion was stimulated by infusing 5 mM L-arginine hydrochloride Sigma ; , 1 nM VIP Peninsula Laboratories Europe ; , or 50 M carbachol Sigma ; and by decreasing perfusate glucose concentration from 11 to 3.2 mM. Integrity of the isolated pancreas preparation was verified by measuring inhibition of glucagon secretion while increasing perfusate glucose concentration from 3.2 to 7 mM. Glucagon was analyzed by RIA 4 ; . Anti-glucagon serum Unger's O4A ; was kindly donated by R. H. Unger Health Sciences Center, University of Texas, Dallas, TX ; . All samples for each series of experiments were analyzed within the same assay. For all analyses, results are expressed as means SE. Integrated hormone responses were calculated using the trapezoidal method as the integrated area of the curve, for specified epochs, above or below the mean preinfusion level average of all the baseline levels for each perfusion ; . The normal distribution of data was tested with the KolmogorovSmirnov test 27 ; . Pairwise comparisons were performed using Student's t-test for unpaired samples. P 0.05 was used as the level of statistical significance throughout. Isolated islets. As a further test of potential direct effects of amylin on pancreatic islets, these were isolated from male Lewis rats Harlan Sprague Dawley, Indianapolis, IN ; weighing between 150 and 200 g. Islets of Langerhans were isolated from whole pancreas by use of a method of collagenase digestion originally described by Kostianovsky et al. 12 ; and modified by Lakey et al. 13 ; . Briefly, the pancreas was dissected free of fat, cleaned, and placed into 5 ml of collagenase-P 1.8 mg ml; Boehringer Mannheim ; plus DNase 0.1 mg ml ; , minced, and incubated at 37C for 20 min. Digested pancreas was then shaken vigorously and washed in HEPES buffer, and islets were separated on a Ficoll gradient. Selected islets were then hand picked and cultured in RPMI FCS 10% ; for 34 days at 37C until experimentation. After being washed in RPMI media for 1 h and then incubated in Hanks' BSS buffer containing 3 mM glucose for 1 h, triplicate groups of 15 islets were then incubated at 37C in fresh Hanks' buffer for a further hour of treatment, after which media were collected and frozen at 70C until assayed for glucagon. Separate treatments consisted of addition of the following to buffer medium: 3 mM D-glucose control ; , 3 mM D-glucose plus 10 mM L-arginine, 3 mM D-glucose plus 10 mM L-arginine plus 100 nM somatostatin, 3 mM D-glucose plus 10 mM L-arginine plus 100 nM rat amylin and pentostatin.

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Ten baboons Papio anubis ; weighing 4 to 9 were anesthetized with intravenous pentobarbital 30 mg per kilogram body weight. Pentobarbital was supplemented as required to maintain light anesthesia. After tracheostomy gallamine triethiodide Flaxedil ; was administered in doses and phenobarbital.

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