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In my practice, I commonly use radiation therapy to treat the whole spectrum of solid malignant tumors. Radiation therapy is often used after surgery or chemotherapy, as a second stage in treatment. Sometimes, however, radiation therapy is used concurrently with chemotherapy, or even as the first or only modality of treatment. I treat approximately 20 patients each day and provide follow-up care and or consultation with another 5 or so patients a day. I currently have approximately 2, 000 patients in various stages of follow-up to their initial treatment. Most of these are long-term survivors. Because of the nature of some cancers, I must sometimes irradiate large portions of my patients' abdomens. Such patients often experience nau888-929-4367 AmericansForSafeAccess 17
The study was an open comparison between the two antibiotics, following the study design used for the same indication in similar studies.4, 12, 13 Patients were randomized to receive one tablet containing 500 mg of clarithromycin MR once a day for 5 days or one tablet containing 590 mg 1 million units ; of penicillin V three times a day for 10 days. To reduce the effects of the open study approach, bacteriological efficacy which was the main efficacy criterion ; was read blindly by the bacteriologist and independently of clinical symptoms. Patients were scheduled for three further visits: a telephone call visit 2 ; on day 3 to confirm that the clinical course was satisfactory and the drug was well tolerated; a visit after treatment visit 3 ; on day 8 1 or depending on the treatment group ; to assess bacteriological and clinical efficacy; and a follow-up visit visit 4 ; on day 30 during which clinical efficacy and tolerability were evaluated. Throat samples obtained at the enrolment visit and visits 3 and 4 were mailed the same day for culture to the central laboratory Laboratoire de Biologie Medicale BIO VSM, Vaires-sur-Marne, France ; using Amies transport medium TGV-AER; Bio-Rad, Marnes-la-Coquette, France ; . Swabs were plated on 5% sheep blood agar bioMrieux, Marcy l'toile, France ; , incubated anaerobically and examined at 24 and 48 h. -Haemolytic streptococcal isolates were typed with group A identification latex reagent bioMrieux ; . Strains of GABHS were tested for susceptibility to penicillin G, erythromycin, clarithromycin and lincomycin by standard disc diffusion test and stored frozen at 70 C. MICs of clarithromycin against susceptible strains were further determined using the NCCLS microdilution procedure. Tolerability was evaluated by reporting all adverse events. An adverse event was defined as any adverse manifestation in a patient who was taking the trial medication, whether or not it was related to the drug, and which occurred while taking the medication or within 30 days of discontinuing the treatment. The investigator evaluated compliance by counting the tablets returned at the post-treatment visit and by reading a diary in which each patient recorded medication administration. The Dijon Advisory Committee for the Protection of Persons in Biomedical Research approved the protocol for this study. The study was conducted in accordance with the ethics principles of the Declaration of Helsinki. Each patient or both parents of a minor patient ; signed a free, informed, written consent during the first visit of the trial.
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ASP was able to induce more persistent ASP activity and a faster rate of complete hematologic remission.10 Most of the studies performed in Europe with PEG-ASP have been conducted in the frame of BFM ALL frontline or relapsed protocols.12, 27-29 One study was performed within the ALL BFM 95 protocol to determine whether one single dose 1, 000 U m2 i.v. ; of Medac Oncaspar administered during reinduction protocol II, second exposure to ASP ; could reduce the 30% rate of clinical allergic reactions expected to happen in that phase with the use of the native E. coli ASP. The study also sought to maintain the target ASP activity levels of 100 U L and thus adequate serum asparagine depletion ; for 2 weeks and 50 U L for 3 weeks thus comparable to those obtained with four doses of the native E. coli ASP product given at a dose of 10, 000 U m2 ; . allergic reactions were observed during the reinduction phase protocol II ; among the 66 investigated children but the target ASP activity level of 100 U L was maintained for 14 days in only about 70% of the patients, with a rapid decline of ASP activity levels after 14 days in about 30% of patients, most probably because of the onset of silent inactivation.12 Our study was designed to evaluate the pharmacokinetic and pharmacodynamic behavior of PEG-ASP given at the dosage of 1, 000 U m2 in the induction and reinduction phases of a BFM-based treatment to evaluate whether this schedule could provide comparable treatment intensity to that of the native E. coli ASP product and avoid the early and fast fall of plasma enzymatic activity silent inactivation ; observed by Mller et al.12 In our study, serum ASP activity levels were monitored during induction protocol I ; for 15 days after the first dose days 1227 ; , for 18 days after the second dose days 2745 ; and during reinduction protocols II or III ; for 23 days days 123 ; to evaluate the activity timecourse of the schedule adopted. In order to evaluate whether our patients had received a treatment intensity comparable to that provided by a treatment schedule including the native E. coli ASP product, we compared data derived from pharmacokinetic monitoring on the use of the native E. coli ASP given in induction protocol I ; of the BFM ALL 90 study at the dosage of 5, 000 U m2 i.v. every 3 days8 this schedule is currently used in the front-line induction treatment of the AIEOP ALL 2000 study ; . Mean trough enzyme activities measured before the next ASP dose, given every 3 days ; averaged 270109 U L median 265 U L, range 85-552 U L ; with 95.9% of samples completely i.e.0.1 M ; and 4.1% almost completely 0.1 and 0.5 M ; depleted of plasma asparagine, with this depletion persisting for 9 additional days i.e. until day 42 ; after the last dose day 33 ; .29 In our study ASP activity levels were 100 U L during induction at almost every sampling point, with the exception of samples from three patients on days 27, 39 and 45; although ASP activity levels in these 9 samples were below 100 U L, they were 70 U L eight patients and 31.9 U L in only one case. Interestingly the plasma asparagine levels were below the detection limit i.e. 0.2 M ; in all samples. During reinduction second exposure ; one dose 1, 000 U m2 i.v. ; of PEG-ASP was used in our study at the same dosage as that used in.
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Tion reactions of peroxynitrite. Biochem Biophys Res Commun 285: 782787, 2001. Schroeder P, Zhang H, Klotz LO, Kalyanaraman B, Sies H. ; -Epicatechin inhibits nitration and dimerization of tyrosine in hydrophilic as well as hydrophobic environments. Biochem Biophys Res Commun 289: 1334 1338, Schulz JB, Matthews RT, Klockgether T, Dichgans J, Beal MF. The role of mitochondrial dysfunction and neuronal nitric oxide in animal models of neurodegenerative diseases. Mol Cell Biochem 174: 193197, 1997. Schulz R, Dodge KL, Lopaschuk GD, Clanachan AS. Peroxynitrite impairs cardiac contractile function by decreasing cardiac efficiency. J Physiol Heart Circ Physiol 272: H1212H1219, 1997. Schuman EM, Madison DV. A requirement for the intercellular messenger nitric oxide in long-term potentiation. Science 254: 15031506, 1991. Schutte H, Mayer K, Burger H, Witzenrath M, Gessler T, Seeger W, Grimminger F. Endogenous nitric oxide synthesis and vascular leakage in ischemic-reperfused rabbit lungs. J Respir Crit Care Med 164: 412 418, Schutte H, Witzenrath M, Mayer K, Rosseau S, Seeger W, Grimminger F. Short-term "preconditioning" with inhaled nitric oxide protects rabbit lungs against ischemia-reperfusion injury. Transplantation 72: 13631370, 2001. Scott DJ, Hull MA, Cartwright EJ, Lam WK, Tisbury A, Poulsom R, Markham AF, Bonifer C, Coletta PL. Lack of inducible nitric oxide synthase promotes intestinal tumorigenesis in the Apc Min ; mouse. Gastroenterology 121: 889 899, Scott GS, Hooper DC. The role of uric acid in protection against peroxynitrite-mediated pathology. Med Hypotheses 56: 95100, 2001. Scott GS, Kean RB, Southan GJ, Szabo C, Hooper DC. Effect of mercaptoethylguanidine scavengers of peroxynitrite on the development of experimental allergic encephalomyelitis in PLSJL mice. Neurosci Lett 311: 125128, 2001. Sekar K. Inhaled nitric oxide in term and preterm infants. J Perinatol 26 Suppl 1: S4 S23, 2006. Seki J, Nishio M, Kato Y, Motoyama Y, Yoshida K. FK409, a new nitric-oxide donor, suppresses smooth muscle proliferation in the rat model of balloon angioplasty. Atherosclerosis 117: 97 106, Senftleben U, Cao Y, Xiao G, Greten FR, Krahn G, Bonizzi G, Chen Y, Hu Y, Fong A, Sun SC, Karin M. Activation by IKKalpha of a second, evolutionary conserved, NF-kappa B signaling pathway. Science 293: 14951499, 2001. Serracino-Inglott F, Virlos IT, Habib NA, Williamson RC, Mathie RT. Differential nitric oxide synthase expression during hepatic ischemia-reperfusion. J Surg 185: 589 595, Serrano F, Kolluri NS, Wientjes FB, Card JP, Klann E. NADPH oxidase immunoreactivity in the mouse brain. Brain Res 988: 193198, 2003. Shacka JJ, Sahawneh MA, Gonzalez JD, Ye YZ, D'Alessandro TL, Estevez AG. Two distinct signaling pathways regulate peroxynitrite-induced apoptosis in PC12 cells. Cell Death Differ 13: 1506 1514, Shah AM, MacCarthy PA. Paracrine and autocrine effects of nitric oxide on myocardial function. Pharmacol Ther 86: 49 86, Shall S, de Murcia G. Poly ADP-ribose ; polymerase-1: what have we learned from the deficient mouse model? Mutat Res 460: 115, 2000. Sharma VS, Traylor TG, Gardiner R, Mizukami H. Reaction of nitric oxide with heme proteins and model compounds of hemoglobin. Biochemistry 26: 38373843, 1987. Shears LL 2nd, Kibbe MR, Murdock AD, Billiar TR, Lizonova A, Kovesdi I, Watkins SC, Tzeng E. Efficient inhibition of intimal hyperplasia by adenovirus-mediated inducible nitric oxide synthase gene transfer to rats and pigs in vivo. J Coll Surg 187: 295306, 1998. Sheng H, Enghild JJ, Bowler R, Patel M, Batinic-Haberle I, Calvi CL, Day BJ, Pearlstein RD, Crapo JD, Warner DS. prv.
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Izumi Z, Covey T, Crouse J, Garcia A, Xu W, Del Castillo J, Biron J, Cole S, Hu MC-T, Pacifici R, Ponting I, Saris C, Wen D, Yung YP, Lin H, Bosselman RA. Identification and cloning of a megakaryocyte growth and development factor that is a ligand for the cytokine receptor Mpl. Cell. 1994; 77: 1117-1124. Kato T, Ogami K, Shimada Y, Iwamatsu A, Sohma Y, Akahori H, Horie K, Kokubo A, Kudo Y, Maeda E. Purification and characterization of thrombopoietin. J Biochem. 1995; 118: 229-236. Zeigler FC, de Sauvage F, Widmer HR, Keller GA, Donahue C, Schreiber RD, Malloy B, Hass P, Eaton D. Matthews W. In vitro megakaryocytopoietic and thrombopoietic activity of c-Mpl ligand TPO ; on purified murine hematopoietic stem cells. Blood. 1994; 84: 4045-4052. Debilit N, Wendling F, Katz A, Guichard J, Breton-Gorius J, Hunt P, Vainchenker W. The Mpl-ligand or thrombopoietin or megakaryocyte growth and differentiative factor has both direct proliferative and differentiative activities on human megakaryocyte progenitors. Blood. 1995; 86: 2516-2525. Nichol JL, Hokom MM, Homkohl A, Sheridan WP, Ohashi H, Kato T, Li YS, Bartley TD, Choi E, Bogenberger J, Skrine JD, Knudten A, Chen J, Trail G, Sleeman L, Cole S, Grampp G, Hunt P. Megakaryocyte growth and development factor. Analyses of in vitro effects on human megakaryopoiesis and endogenous serum levels during chemotherapy-induced thrombocytopenia. J Clin Invest. 1995; 95: 2973-2978. Kaushansky K, Broudy VC, Lin N, Jorgensen MJ, McCarty J, Fox N, Zucker-Franklin D, Lofton-Day C. Thrombopoietin, the Mpl ligand, is essential for full megakaryocyte development. Proc Natl Acad Sci USA. 1995; 92: 3234-3238.
The scrub typhus group of rickettsiae contains one species Rickettsia tsutsugamushi ; consisting of a multitude of strains which exhibit serologically distinct epitopes as well as many cross-reactive epitopes. These obligate intracellular bacteria, which are transmitted to humans by infected mites, are the causative agents of scrub typhus fever. The disease is endemic in many areas of the Orient, including Japan, Southeast Asia, and the Philippines. Structurally, scrub typhus rickettsiae resemble gram-negative bacteria, in that distinct outer and inner membranes are present 15 ; . Conponents of the envelope, such as proteins, lipids, and carbohydrates, are largely undefined owing to the technical difficulties in obtaining large quantities of scrub typhus rickettsiae for analysis. Recently; several different proteins, with a variety of molecular weights, have been identified as R. tsutsugamushi structural components. A subset of these proteins is recognized by sera obtained from immunized animals 3, 6, 7, ; and infected humans unpublished observations ; . Although analytical approaches can be used to study scrub typhus protein antigens, it still remains technically and economically unfeasible to produce large quantities of purified R. tsutsugamushi for the isolation of individual antigens for vaccine purposes. Expression of rickettsial antigens via recombinant DNA technology should overcome this problem to a large degree. This report describes the cloning of R. tsutsugamushi genes for two protein antigens in the Xgtll expression vector system. This recombinant system, which has been used successfully for cloning several antigens of other pathogens which are difficult to grow 19, 22 ; , allowed us to minimize potential problems with the expression of rickettsial genes and antigen instability. We report the successful cloning of the 110 kilodalton 110K ; and 56K polypeptide antigens of R. tsutsugamushi Karp; we demonstrate with antibody, affinity purified against the recombinant antigens, that crossreactive antigens of similar molecular weights are present in the Kato and Gilliam strains as well and kava.
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27. Vallon V, Lang F. New insights into the role of serum- and glucocorticoid-inducible kinase SGK1 in the regulation of renal function and blood pressure. Curr Opin Nephrol Hypertens. 2005; 14: 59 Friedrich B, Warntges S, Klingel K, Sauter M, Kandolf R, Risler T, Muller GA, Witzgall R, Kriz W, Grone HJ, Lang F. Up-regulation of the human serum and glucocorticoid-dependent kinase 1 in glomerulonephritis. Kidney Blood Press Res. 2002; 25: 303307. de Kloet ER, Van Acker SA, Sibug RM, Oitzl MS, Meijer OC, Rahmouni K, de Jong W. Brain mineralocorticoid receptors and centrally regulated functions. Kidney Int. 2000; 57: 1329 Schiffrin EL. The many targets of aldosterone. Hypertension. 2004; 43: 938 Wolf G, Chen S, Ziyadeh FN. From the periphery of the glomerular capillary wall toward the center of disease: podocyte injury comes of age in diabetic nephropathy. Diabetes. 2005; 54: 1626 Leong ML, Maiyar AC, Kim B, O'Keeffe BA, Firestone GL. Expression of the serum- and glucocorticoid-inducible protein kinase, Sgk, is a cell survival response to multiple types of environmental stress stimuli in mammary epithelial cells. J Biol Chem. 2003; 278: 58715882. Quinkler M, Zehnder D, Eardley KS, Lepenies J, Howie AJ, Hughes SV, Cockwell P, Hewison M, Stewart PM. Increased expression of mineralocorticoid effector mechanisms in kidney biopsies of patients with heavy proteinuria. Circulation. 2005; 112: 14351443. Kumar JM, Brooks DP, Olson BA, Laping NJ. Sgk, a putative serine threonine kinase, is differentially expressed in the kidney of diabetic mice and humans. J Soc Nephrol. 1999; 10: 2488 Vallon V, Huang DY, Grahammer F, Wyatt AW, Osswald H, Wulff P, Kuhl D, Lang F. SGK1 as a determinant of kidney function and salt intake in response to mineralocorticoid excess. J Physiol Regul Integr Comp Physiol. 2005; 289: R395R401. 36. Nagata K, Obata K, Xu J, Ichihara S, Noda A, Kimata H, Kato T, Izawa H, Murohara T, Yokota M. Mineralocorticoid receptor antagonism attenuates cardiac hypertrophy and failure in low-aldosterone hypertensive rats. Hypertension. 2006; 47: 656 Farjah M, Roxas BP, Geenen DL, Danziger RS. Dietary salt regulates renal SGK1 abundance: relevance to salt sensitivity in the Dahl rat. Hypertension. 2003; 41: 874 Hou J, Speirs HJ, Seckl JR, Brown RW. Sgk1 gene expression in kidney and its regulation by aldosterone: spatio-temporal heterogeneity and quantitative analysis. J Soc Nephrol. 2002; 13: 1190 Walker WG. Hypertension-related renal injury: a major contributor to end-stage renal disease. J Kidney Dis. 1993; 22: 164
Statistical analyses Demographic data were analysed using Student's t-test for continuous variables and 2-test for categorical variables. Analyses of covariance ANCOVAs ; were performed for the HADS scores, the HSCL scores and the SSQS scores, while controlling for the baseline scores. For the analysis of the DRK scores, a distinction was made between seven individual IVF treatment stages: baseline, ovarian stimulation, oocyte retrieval, fertilization, embryo transfer, waiting period and pregnancy test. Stage scores for both positive and negative affect were calculated by averaging daily scores on the DRK within each treatment stage. ANCOVAs were conducted for group comparisons of both positive and negative affect during each individual treatment stage, adjusting for baseline affect scores. Analyses for the day of the pregnancy test were also statistically controlled for pregnancy outcome. To determine changes in affect over treatment for all subjects, including women whose first IVF cycle was cancelled, random effects regression analysis was conducted. Random effects regression allows for missing observations, assessments at different end-points, timeindependent co-variables and time-dependent co-variables. Individual time trend curves are based on the available data from a specific individual and data from all other subjects: intercepts represent estimated baseline functioning, while slopes characterize change in functioning over time. Since affect is strongly related to treatment outcome, the first treatment cycle was divided into periods: the period before treatment outcome was known from baseline until the waiting days ; and the day that treatment outcome became known. In the random effects regression analyses for stage scores from baseline until the waiting period, both dependent variables negative affect and positive affect ; were modelled on the basis of a random intercept term, a random effect representing time stage ; in treatment, and fixed effects representing treatment mild IVF versus conventional IVF ; and cancellation yes versus no ; . In second series of random effects regression analyses, both dependent variables were modelled on the basis of a random intercept term, a random effect representing time before versus after treatment outcome ; in treatment, and fixed effects representing treatment mild IVF versus conventional IVF ; , pregnancy no versus yes ; and cancellation no versus yes ; . Interaction terms were entered into the models if it made sense both clinically and statistically. All models were adjusted for the time-independent co-variable hospital Rotterdam versus Utrecht ; and were fitted using restricted maximum likelihood measures. The covariance matrix was specified as unstructured general covariance ; . All analyses were performed using the Statistical Package for the Social Sciences SPSS version 10.1 ; , while random effects regression models were implemented with the PROC MIXED procedure of the SAS System version 8.2 ; . Significance testing on all outcome measures was done at the 0.05 level of significance two-tailed ; . Effect sizes were measured using Cohen's d Cohen, 1988 ; . The SD of the conventional IVF group was used as the denominator of Cohen's d and keppra.
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Roberto Pili, M.D. 88. * Wang XF, Qian DZ, Ren M, Kato Y, Wei Y, Zhang L, Fansler Z, Clark D, Nakanishi O, Pili R. Epigenetic modulation of retinoic acid receptor beta2 by the histone deacetylase inhibitor MS-275 in human renal cell carcinoma. Clin Cancer Res. 2005 ; 11 9 ; , 3535. 89. * Qian DZ, Ren M, Wei Y, Wang X, van de Geijn F, Rasmussen C, Nakanishi O, Sacchi N, Pili R. In vivo imaging of retinoic acid receptor beta2 transcriptional activation by the histone deacetylase inhibitor MS-275 in retinoid-resistant prostate cancer cells. Prostate. 2005 ; 64 1 ; , 20. Elizabeth Platz, Sc.D. 90. * Platz EA, Rohrmann S, Pearson JD, Corrada MM, Watson DJ, De Marzo AM, Landis PK, Metter EJ, Carter HB. Nonsteroidal anti-inflammatory drugs and risk of prostate cancer in the Baltimore Longitudinal Study of Aging ncer Epidemiol Biomarkers Prev. 2005 ; 14 2 ; , 390. 91. * Platz EA, Pollak MN, Leitzmann MF, Stampfer MJ, Willett WC, Giovannucci E. Plasma insulin-like growth factor-1 and binding protein-3 and subsequent risk of prostate cancer in the PSA era. Cancer Causes Control. 2005 ; 16 3 ; : 255. 92. * Parsons JK, Carter HB, Platz EA, Wright EJ, Landis P, Metter EJ. Serum testosterone and the risk of prostate cancer: potential implications for testosterone therapy. Cancer Epidemiol Biomarkers Prev. 2005 ; 14 9 ; , 2257. Ingo Ruczinski, Ph.D. 93. * Kooperberg C, Ruczinski I. Identifying interacting SNPs using Monte Carlo logic regression. Genet Epidemiol. 2005 ; 28 2 ; , 157. Charles Rudin, M.D. 94. * Rudin CM, Marshall JL, Huang CH, Kindler HL, Zhang C, Kumar D, Gokhale PC, Steinberg J, Wanaski S, Kasid UN, Ratain MJ. Delivery of a liposomal c-raf-1 antisense oligonucleotide by weekly bolus dosing in patients with advanced solid tumors: a phase I study. Clin Cancer Res. 2004 ; 10 21 ; , 7244. Keerti Shah, Ph.D. and Stephen Yang, M.D. 95. * Rollison DE, Helzlsouer KJ, Halsey NA, Shah KV, Viscidi RP. Markers of past infection with simian virus 40 SV40 ; and risk of incident non-Hodgkin lymphoma in a Maryland cohort. Cancer Epidemiol Biomarkers Prev. 2005 ; 14 6 ; , 1448.
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This report was reviewed for medical and scientific accuracy by James M. Oleske, MD, MPH, Francois-Xavier Bagnoud Professor of Pediatrics, Director, Division of Pulmonary, Allergy, Immunology & Infectious Diseases, Department of Pediatrics, University of Medicine & Dentistry of New Jersey--New Jersey Medical School, Newark, New Jersey and ketoprofen.
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4. Diamandis EP, Yousef GM, Soosaipillai AR, Bunting P. Human kallikrein 6 zyme protease M neurosin ; : a new serum biomarker of ovarian carcinoma. Clin Biochem 2000; 33: 579 Luo LY, Bunting P, Scorilas A, Diamandis EP. Human kallikrein 10: a novel tumor marker for ovarian carcinoma? Clin Chim Acta 2001; 306: 111 Diamandis EP, Okui A, Mitsui S, Luo LY, Soosaipillai A, Grass L, et al. Human kallikrein 11: a new biomarker of prostate and ovarian carcinoma. Cancer Res 2002; 62: 295300. Diamandis EP, Yousef GM. Human tissue kallikrein gene family: a rich source of novel disease biomarkers. Expert Rev Mol Diagn 2001; 1: 18290. Diamandis EP, Yousef GM, Clements J, Ashworth LK, Yoshida S, Egelrud T, et al. New nomenclature for the human tissue kallikrein gene family. Clin Chem 2000; 46: 1855 Yoshida S, Taniguchi M, Hirata A, Shiosaka S. Sequence analysis and expression of human neuropsin cDNA and gene. Gene 1998; 213: 9 Chen ZL, Yoshida S, Kato K, Momota Y, Suzuki J, Tanaka T, et al. Expression and activity-dependent changes of a novel limbicserine protease gene in the hippocampus. J Neurosci 1995; 15: 5088 Okabe A, Momota Y, Yoshida S, Hirata A, Ito J, Nishino H, et al. Kindling induces neuropsin mRNA in the mouse brain. Brain Res 1996; 728: 116 Momota Y, Yoshida S, Ito J, Shibata M, Kato K, Sakurai K, et al. Blockade of neuropsin, a serine protease, ameliorates kindling epilepsy. Eur J Neurosci 1998; 10: 760 Komai S, Matsuyama T, Matsumoto K, Kato K, Kobayashi M, Imamura K, et al. Neuropsin regulates an early phase of Schaffercollateral long-term potentiation in the murine hippocampus. Eur J Neurosci 2000; 12: 1479 Shimizu-Okabe C, Yousef GM, Diamandis EP, Yoshida S, Shiosaka S, Fahnestock M. Expression of the kallikrein gene family in normal and Alzheimer's disease brain. Neuroreport 2001; 12: 274751. Underwood LJ, Tanimoto H, Wang Y, Shigemasa K, Parmley TH, O'Brien TJ. Cloning of tumor-associated differentially expressed gene-14, a novel serine protease overexpressed by ovarian carcinoma. Cancer Res 1999; 59: 44359. Magklara A, Scorilas A, Katsaros D, Massobrio M, Yousef GM, Fracchioli S, et al. The human KLK8 neuropsin ovasin ; gene: identification of two novel splice variants and its prognostic value in ovarian cancer. Clin Cancer Res 2001; 7: 806 Mitsui S, Tsuruoka N, Yamashiro K, Nakazato H, Yamaguchi N. A novel form of human neuropsin, a brain-related serine protease, is generated by alternative splicing and is expressed preferentially in human adult brain. Eur J Biochem 1999; 260: 62734. Luo LY, Grass L, Howarth DJ, Thibault P, Ong H, Diamandis EP. Immunofluorometric assay of human kallikrein 10 and its identification in biological fluids and tissues. Clin Chem 2001; 47: 237 Christopoulos TK, Diamandis EP. Enzymatically amplified timeresolved fluorescence immunoassay with terbium chelates. Anal Chem 1992; 64: 342 Yu H, Diamandis EP. Ultrasensitive time-resolved immunofluorometric assay of prostate-specific antigen in serum and preliminary clinical studies. Clin Chem 1993; 39: 2108 Shimizu C, Yoshida S, Shibata M, Kato K, Momota Y, Matsumoto K, et al. Characterization of recombinant and brain neuropsin, a plasticity-related serine protease. J Biol Chem 1998; 273: 11189 Diamandis EP, Yousef GM, Soosaipillai AR, Grass L, Porter A, Little S, et al. Immunofluorometric assay of human kallikrein 6 and kineret.
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Ishizuka N, see Yamamoto N Isla D, see Hitt R Isoyama K, see Igarashi S Issa B, see Jayson GC Issa J-PJ, Gharibyan V, Cortes J, Jelinek J, Morris G, Verstovsek S, Talpaz M, Garcia-Manero G, Kantarjian HM. Phase II Study of Low-Dose Decitabine in Patients With Chronic Myelogenous Leukemia Resistant to Imatinib Mesylate, 3948 Issell B, see Ezzo J Ito M, see Okada K Itoi E, see Okada K Ivers H, see Savard J Ives N, see Hancock B Ivy SP, see Rubinstein LV Iwao-Koizumi K, Matoba R, Ueno N, Kim SJ, Ando A, Miyoshi Y, Maeda E, Noguchi S, Kato K. Prediction of Docetaxel Response in Human Breast Cancer by Gene Expression Profiling, 422 Iyer R, see Hricak H Izumiyama K, see Onozawa M Izzo B, see Soverini S Izzo F, Ascierto PA J Jablons DM, see Hansen EK Jack AS, see Johnson PWM Jackisch C, see von Minckwitz G Jackson A, see Jayson GC Jackson DV, see Haller DG Jackson E, see Liu G Jackson G, see Tauro S Jackson M, see Rischin D Jacky E, see Breems DA Jacob J-H, see Conroy T see Ducreux M Jacobs I. Screening for Familial Ovarian Cancer: The Need for WellDesigned Prospective Studies editorial ; , 5443 Jacobs IJ, see Menon U see Rockall AG Jacobs MI, see Mantha S Jacobs MS, see Schuch G Jacobsen AB, see Hjermstad MJ Jacobsen PB, see Andrykowski MA Jacobsohn DA, see Bolanos-Meade J ~ Jacobson F, see Travis WD Jacobson JO, see Neuss MN Jacobson JS, see Doyle JJ see Hershman D Jacques DP, see Allen PJ Jaeck D, see Domont J Jaeckle K, see Blaney SM see Galanis E Jaeger U, see Raderer M Jager U, see Kienle DL Jagsi R, DeLaney TF, Donelan K, Tarbell NJ. In Reply correspondence ; , 2438 Jahan TM, see Hansen EK Jahanzeb M, see Neuss MN Jahnke K, see Budach V see Shankier TN Jain RK, see Willett CG Jain V, see Horning SJ Jakesz R, see Filipits M Jaklitsch MT, see Mamon HJ and kato.
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