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Feeling trapped and broken I feel but nothing is spoken Four walls hold me in When will I ever win Same things same routine But never will my mind be clean Scars, pain, and broken memories Someone could you find the keys Unlock me let me go Just please let me know Who I and where I'll be Lemme' know so I don't flee It feels like I'm being crushed Walls closing down soon I'll be dust Lost and confused Just misplaced and used Please don't leave me here Just hold me and make it all disappear Gone and going far away Somehow make it my last day. -- Ashlee S.
Plant materials and culture media: Cymbidium `Tim Hot' shoots 4 cm length ; , derived from protocorm-like bodies PLBs ; cultured on MS Murashige and Skoog, 1962 ; medium containing 0.5 mgl-1 -naphthaleneacetic acid NAA ; , 2 mgl-1 6-benzyladenine BA ; , 1 gl-1 activated charcoal AC ; , 20% coconut water CW ; , 30 gl-1 sucrose and 8 gl-1 agar Haiphong Co., Vietnam ; , were cultured on MS medium containing 0.5 mgl-1 NAA, 1 gl-1 AC, 10% CW, 25 gl-1 sucrose and 8 gl-1 agar. Lilium longiflorum bulb scales, derived from in vitro bulblets cultured on MS medium containing 0.2 - 0.5 mgl-1 BA, 30 gl-1 sucrose and 8 gl-1 agar, were cultured on MS medium supplemented with 1 gl-1 AC, 30 gl-1 sucrose and 8 gl-1 agar. Fragaria vesca cv. `My Da' shoots 1.5 cm ; , derived from meristems cultured on MS medium containing vitamin B5, 0.2 mgl-1 BA, 30 gl-1 sucrose and 8 gl-1 agar, were cultured on MS medium supplemented with 1 gl-1 AC, 30 gl-1 sucrose and 8 gl-1 agar. For all experiments, explants were cultured in vessels 500 ml ; containing 60 ml medium. pH of media was adjusted to 5.7 before autoclaving at 121oC, 1 atm for 40 min. Lighting systems: Cool white fluorescent lamps Neon tubes ; 40 W each; Rang Dong Light source and Vacuum Flask Co., Vietnam, FL-40W T10 ; and warm white fluorescent lamps Compact 3U lamps ; 18 W each; Rang Dong Light source and Vacuum Flask Co., Vietnam, CFH-3U18W ; were used as lighting sources in each experiment. Irradiances were 45 molm-2s-1 under Neon light or 45, 60, 75 molm-2s-1 for Compact 3U system according to each experiment. Photosynthetic photon flux density PPFD ; was measured with.
This day is called the feast of Crispian: He that outlives this day and comes safe home, Will stand a tip-toe when the day is named, And rouse him at the name of Crispian. He that shall live this day and see old age, Will yearly on the vigil feast his neighbours, And say "To-morrow is Saint Crispian": Then will he strip his sleeve and show his scars, And say "These wounds I had on Crispin's day." Old men forget: yet all shall be forgot, But he'll remember with advantages What feats he did that day: then shall our names, Familiar in his mouth as household words, Harry the King, Bedford and Exeter, Warwick and Talbot, Salisbury and Gloucester, Be in their flowing cups freshly remember'd. This story shall the good man teach his son; And Crispin Crispian shall ne'er go by, From this day to the ending of the world, But we in it shall be remember'd; We few, we happy few, we band of brothers; For he today that sheds his blood with me Shall be my brother; be he ne'er so vile, This day shall gentle his condition: And gentlemen in England now a-bed Shall think themselves accursed they were not here, And hold their manhoods cheap whiles any speaks That fought with us upon Saint Crispin's day.
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Drug Activated Charcoal Adenosine Adenocard ; Albuterol Amiodarone Concentration 25 Gm bottle 6 mg 2ml 2.5 mg 3ml NS 150mg 3cc Standard Dosage 1 gm kg not to exceed 50 Gm ; SVT: 0.1 mg kg max 6mg ; . If no response, 0.2 mg kg max 12 mg ; 2.5 mg 3cc NS Pulseless Arrest: 5mg kg IV IO followed by, or diluted in, 20 to 30 ml Max single dose 300mg Tachycardia with Poor Perfusion: 5mg kg IV IO over 20-60 minutes Physician Consult only ; Bradycardia and pretreatment for Pediatric Intubations: 0.02 mg kg IV IO Minimum dose is 0.1 mg or 1ml; single max dose for child is 0.5mg or 5 ml and for adolescents is 1mg or 10ml ; , MR x 1 Organophosphate Poisoning: 0.02 mg kg IV IO; MR q 2-5 min. until drying of secretions Newborn: 2ml kg IV IO Neonatal 3 months: 3ml kg IV IO months - 2yrs: 2ml kg IV IO 2yrs: 1ml kg IV IO 0.1 mg kg 0.02 ml kg ; IV slowly; MR x 2 q min. to max. dose of 0.3 mg kg 0.06 ml kg ; or 0.5 mg kg 0.1 ml kg Rectal max 10 mg 1 mg kg IV IO or max of 50 mg; IV IO max 25mg min Allergic reaction moderate severe anaphylaxis: 0.01 mg kg SQ 0.01 ml kg ; max. of 0.3 mg 0.3 ml ; or Pedi Auto-Injector; repeat prn in 5 minutes Anaphylaxis: if no response to Epi 1: 1000, give 1: 10, 000 0.01 mg kg 0.1 ml kg ; IV Bradycardia: 0.01 mg kg 0.1 ml kg ; IV Cardiac Arrest: 0.1 ml kg IV.
WE had a really great day and would like more!" was the clear message from customers who attended Derwent Living's first customer induction day. Resident involvement officer Sue Williams arranged the day to bring customers up to date with what was happening at Derwent Living. Sue said: "A spring 2006 research project told us that some customers felt Derwent Living had grown so quickly, they no longer knew who was who or what was what so we arranged a day for customers to come and meet us." Customers reported that they found the tour of the building interesting, the ID parade great fun and they wanted the speed networking to last longer as they learned so much about "what makes Derwent tick." Staff and customers socialised over a buffet lunch and the afternoon included "a day in the life of a customer service advisor" and learning why Derwent Housing became Derwent Living. Sue said: "My favourite bit was the question time session. I heard about the famous people our customers had met, staff who had made them laugh and that if Derwent Living were an animal, they thought it would be an octopus!" The day last October was so popular that we plan to put on two further customer days in 2007. To reserve a place, or to find out more, please contact Sue Williams, resident involvement officer on 01332 614825.
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Within the framework of the Zambian study of the project, the energy component is intended to give inputs in the following broad objectives: i ; To find ways of including forest management as a part of charcoal production ii ; To improve the understanding of how, why and where charcoal production areas are established and who is involved in charcoal production iii ; To propose interventions that can be carried out in order to: a ; secure the supply of charcoal to low-income households, and b ; mitigate adverse effects of charcoal production iv ; To outline different scenarios of fuel utilisation under different policy options v ; To inform policy makers, energy analysts, forest officers and other national and international bodies about the research and its outcome. Against these broad objectives and others that relate to the ecological aspects of the study, the component aims to perform the following tasks: Conduct participatory surveys in selected charcoal production areas with different characteristics. Determine the relationships between production areas, transport systems and retailing outlets. Identify and map land tenure systems and land use in present and potential charcoal production areas. Further, analyse the implications on charcoal production depending on land use and tenure system. Analyse the implication of charcoal production in relation to population demographics, employment and economic development Estimate trends in urban charcoal demand under various assumptions Investigate the extent to which charcoal production is separated from agricultural activities or integrated with them and the existence of professional, full-time charcoal producers and their access to land for charcoal production and their interaction with the local population. 1.2. Approaches and Methodologies Used and chlorambucil.
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Competition between Hzfolate and Methotrexate for binding to Hzfolate Reductase at O"C-Methotrexate binds very tightly to Hzfolate reductase in the presence of excess NADPH 24 ; . To determine the effect of Hzfolate on this high affinity [3H]methotrexate binding under conditions in which reduction of H2folate cannot occur, studies were performed at 0C. [3H]Methotrexate and varying concentrations of folate or H, folate were premixed at 0C then a chilled solution containing Hzfolate reductase 0.6 nM ; and NADPH 100 ; was added and the mixture was held at 0C for 5 to 10 min. Bound ["Hlmethotrexate was then assayed by the charcoal adsorption procedure as described under "Materials and Methods." Fig. 1 shows the effect of Htfolate on [3H]methotrexate binding over a wide range of Hzfolate concentrations. In this representative experiment, inhibition of ["Hlmethotrexate binding by HZfolate is first observed at approximately 1 H?folate, a concentration in the range of the Hzfolate Km for this enzyme 13 ; , and is nearly complete at 20 Hzfolate, a concentration which has been reported for intracellular Hzfolate after exposure of L1210 cells to methotrexate 4 ; . Folic acid by contrast does not significantly affect ["Hlmethotrexate binding at pH 7.4 at concentrations below 100 pM. Hzfolate binding is slowly reversible at 0C since incubation of enzyme with Hzfolate and methotrexate for long periods 0C 1 h more ; results in increased binding of methotrexate indicating displacement of bound Hzfolate by methotrexate. Incubation at 0C of the preformed [3H]methotrexate complex with Hzfolate, excess unlabeled methotrexate, or with bovine serum albumin-charcoal for 1 h did not result in a significant decrease in bound [3H]methotrexate indicating that the offrate for methotrexate at this temperature is negligible and that the effects of Hzfolate are solely based on competition.
In our laboratory. Briefly, celite-545 was with 6N HCI followed by distilled water and dried in an oven at 1000 F. It was then mixed 2: 1 w with a mixture of ethylene glycol: propylene glycol 1: lv v ; , and the columns were packed with this mixture. Pregnenolone was eluted by using system I. The maximum recovery was 80%. RIA was carried out by diluting the samples in 1: 8 vlv ; . The sensitivity of the assay was 25 pg mI. Dextran coated charcoal was used to separate the free from the bound and chlordiazepoxide.
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To determine whether the HA cells were capable of reconstituting the hemopoietic compartment of a lethally ablated recipient, FACS was used to isolate Lin HA and Lin HA subpopulations and the cells were assayed in vivo using the congenic Ly5.1 Ly5.2 mouse transplant model.26 A total of 5000 to 6000 Lin HA cells reconstituted all hemopoietic lineages in the peripheral blood of lethally irradiated recipients 8 weeks after transplantation Table 1 ; , whereas an equivalent number of Lin HA cells failed to do so, with the recipients dying 14 to 16 days after transplantation. Isolating the more enriched HSC Lin Sca population, and subfractionating on the basis of HA, resulted in reconstitution of multiple hemopoietic lineages of lethally irradiated recipients 4 weeks after transplantation of 500 Lin Sca HA cells Table 1 ; . However, recipients given transplants of 500 Lin Sca HA cells exhibited an average of only 9.4% donor engraftment, which is significantly lower than predicted from infused unfractionated Lin Sca cells. Studies by Spangrude and Scollay demonstrate that transplanting as few as 100 Lin Sca cells results in the survival of greater than a third of recipients with more than 75% donor cells in the peripheral blood 12 weeks after transplantation.27 In only 1 of 6 recipients were HA cells capable of rescuing lethally irradiated recipients following a transplant of 500 Lin Sca HA cells ; , suggesting that fewer long-term repopulating cells LTRCs ; are contained within this population compared with and chlorothiazide.
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Concentration-action curve, type I Fig. 1 ; action curve, type II Fig. 2 ; Adsorption curves of benzoic acid and succinic acid Fig. 3 ; -isotherm of pilocarpine by animal charcoal Fig. 4 ; of nicotine by animal charcoal Fig. 5 ; isotherm l# garithmated ; of pilocarpine by animal charcoal Fig. 6 ; . Strip of cat gut suspended in 75 cc. of Tyrode solution Fig. 7, a-d ; Isolated strip of cat gut suspended in 75 cc. Tyrode solution Fig. 8, a-e ; Amount and concentration of pilocarpine Fig. 9 ; Isolated cat gut suspended in 75 cc. of Tyrode solution Fig. 10, a-c ; Circular maze viewed from above Fig. 1 ; maze with camera lucida attachment Fig. 2 ; Graphs of the secretion of saliva by a dog from 5 mgm. of pilocarpine nitrate injected subcutaneously at P, preceded by hyoscine hydrobromide at H Fig. 1 ; of the secretion of saliva by another dog from 5 mgm. pilocarpine nitrate injected subcutaneously at P, preceded by hyoscine hydrobromide at H Fig. 2 ; Effect of hyoscines on the movements of the surviving bowel in 50 cc. of oxygenated Ringer's solution exp. 1 ; , Fig. 3 ; Tracing of surviving intestine of rabbit in 50 cc. of Tyrode's solution Fig. 4 ; . Records of movements of a piece of surviving intestine of rabbit in Ringer's solution Fig. 5 ; Time factor in solution of tannin-protein compounds Fig. 1 ; factor in solution of tannin-protein compounds Fig. 2 ; factor in hydrolysis of acetannins Fig. 3 ; Length of life in 48-hour periods ; of rabbits given potassium arsenite alone, and of animals given potassium arsenite and later magnesium sulphate Fig. 1 ; Influence of colloids on non-colloidal drugs Fig. 1, a-h ; Action of drugs upon muscular work of frogs Fig. 1 ; of drugs upon muscular work of frogs Fig. 2 ; of drugs upon muscular work of frogs Fig. 3 ; of drugs upon muscular work of frogs Fig. 4 ; of drugs upon muscular work of frogs Fig. 5 ; Control fatigue curve; perfusion with Ringer's solution Fig. 6 ; Fatigue curve; perfusion with Ringer's solution Fig. 7 ; curve; perfusion with Ringer's solution Fig. 8 ; curve; perfusion with Ringer's solution Fig. 9 ; curve; perfusion with Ringer's solution Fig. 10 ; curve; perfusion with Ringer's solution Fig. 11 and chlorpheniramine.
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Should be noted that prior occupation is the legal basis in Canadian law for both aboriginal title and the limited aboriginal rights, which are chiefly fishing and hunting rights. However, the analytical scheme and details for acquiring either title or limited rights differs. The common law foundation of the aboriginal rights, including title, is evident. 1513 First, I should mention that the law related to occupation is undeveloped, because it has simply not been used for centuries. Nonetheless, occupation of lands terra nullius must be seen as the very first method of acquiring rights to specific pieces of land. In fact, it is understood as an independent source of legal acquisition laga fng ; . Normally, this was done through cultivation, as happened in Sweden 1514 . While the Saami were the very first inhabitants in many parts of the vast present reindeer herding area, their possession and use of lands and natural resources must, subsequently, be understood as a peaceful occupation. The possibility of acquiring limited rights through prior occupancy and continuous use of an area is, however, unclear. To my knowledge, it has not been discussed in Swedish literature. Nevertheless, it should be possible 1515 and chlorpromazine.
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Of community-acquired pneumonia.8 The study drug was administered either orally or intravenously as a 1 infusion. At the investigator's discretion, patients received either oral therapy alone, or intravenous therapy followed by oral therapy. In brief, 111 gatifloxacin blood concentrations collected from 67 patients were available for model development. Two blood samples were collected between days 2 and 4, the first taken on arrival at the clinic prior to the dose, and the second taken 2 h post-dose. If the patient could not wait 2 h for the second sample to be collected, only the first was collected. However, the majority of patients had two samples collected 66% ; . The demographics of these 67 patients were mean S.D. ; : age 50 17 years, serum creatinine 0.81 0.21 mg dL, weight 82.5 19.1 kg and estimated creatinine clearance 91.0 30.3 mL min. A one-compartment model, with first order absorption and elimination, was fitted to the data, resulting in the following equation for the prediction of gatifloxacin clearance in this Phase III patient population: Predicted Clj 8.43 + 0.036 CrClj 91.0 ; + 0.073 Wtj 82.5 ; 1 ; where Clj is the typical value of clearance in L h for the jth patient, CrClj is estimated creatinine clearance for the jth patient in mL min and Wt is weight in kg for the jth patient. The following exponential error model described the inter-individual variability in Cl: Clj Clj ej Cl ; 2 ; where j Cl ; is the persistent difference between the true value of the Cl parameter in the jth patient and the predicted value; the j Cl ; are independent, identically distributed statistical errors with a mean of zero and a variance equal to 2x. The population mean Cl expressed as %CV was 22.18 for these data. The predictive performance of this model, stratified by patient age 60 versus 60 years ; , was evaluated by examining summary statistics of the population mean prediction errors and absolute population mean prediction errors. The relationships between Bayesian-estimated and model-predicted clearance, stratified by patient age 60 versus 60 years ; , were evaluated graphically.
Three operational modes of carbonisation can be distinguished: - Carbonisation by partial combustion; - Carbonisation by injection of hot gas in the load; - Carbonisation in an enclosed reactor. 2.1. Partial combustion furnaces For this technique, the energy required for carbonisation is provided by the combustion of part of the load, placed inside a sealed enclosure, which reduces the yield of wood charcoal at the same rate. All condensable products as well as gases are usually not recovered. The transformation of wood into charcoal by this method is poor, since over 50% of the initial energy is lost. Furthermore, the inconvenience of this method is that it does not allow carbonisation of straw, reeds or cotton stems as well as biomass of weak granulometry such as rice husks, coffee husks and sawdust. The main reasons are: Poor thermal transfer in the carboniser load; Appearance of chimneys in the load prevents lateral thermal transfer in the case of straw; Difficulty in controlling air entries, which can potentially lead to total inflammation of the load and chlorpropamide.
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Soil samples were collected in plastic tubes 6415 cm ; from the topsoil: 20 replicates before burning control ; , 20 replicates immediately after the burning burnt ; and 20 replicates one year after burning burnt 1 yr ; , and cut into three depth intervals 01, 12.5, 2.55 cm ; . The samples were dried at 40 C for 24 h and weighed to calculate the bulk density. The aggregates were crushed and coarse material roots and charcoal particles ; 2000 m was separated by sieving. Sub-samples were ball-milled for carbon and nitrogen analyses. Total carbon and nitrogen concentrations were determined for all soil samples by dry combustion via an elemental analyzer Elementar VarioEL ; . The values for total organic carbon corresponded to the total carbon content because the soil samples did not contain bicarbonates. The analysis of charred material in the soil samples was performed using mid infrared Fourier transformed infrared spectroscopy MIR-DRIFT ; Viscarra Rossel et al., 2006; Janik et al., 2007 ; . Samples were grinded and directly measured, and the 599 and charcoal.
2224 Nucleic Acids Research, 2007, Vol. 35, No. 7 hydroxyl oxygens of Mg2-coordinated antibiotic dimers Figure 1 ; and the 2-amino of guanines 13, 16 ; . MTA and MSK contain the same tricyclic core moiety and oligosaccharide side chains, but they differ in the side chain at C-3. This chain is longer in MTA and bears a higher number, and different arrangement, of potential donors and acceptors of hydrogen bonds Figure 1 ; . Hence, a higher more negative ; free binding energy G ; for MTA is consistent with the formation of extra hydrogen bonds. The oligosaccharide moieties of the mithramycins are the same. They are involved in the binding within the DNA minor groove, forming equivalent intermolecular contacts with the sugar-phosphate backbone 4, 13 ; . The higher binding constant determined for MTA is consistent with its capacity to displace Sp1 protein from its putative C G-rich binding sites at concentrations below those at which MSK does this 10 ; . The comparison between MTA and MSK binding to DNAs of diverse C G content Figure 6 ; highlights the fact that small substitutions in naturally occurring DNA-binding molecules have significant effects on intermolecular interactions, which for mithramycin analogues appears to result in different biological properties 20 and chlorzoxazone.
Address for reprint requests and other correspondence: W. M. Chilian, Dept. of Physiology, Medical College of Wisconsin, 8701 Watertown Plank Rd., Milwaukee, WI 53226 E-mail: chilian mcw ; . : ajpheart
Complex formation of rifamycins with RNA 90o polymerase. In studies of the interaction between RNA polymerase and rifamycins, it has been found that, by simply mixing the free enzyme E 70 I with the antibiotic, a complex is formed that can 0 0be isolated by passage through a Sephadex 0 50column 176 ; . In contrast to the DNA-enzyme E complex, the antibiotic-enzyme complex is formed N 30 to the same extent at salt concentrations as high 10. as 1 M. Quantitative measurements have shown that 1 mole of holo enzyme az383'a ; binds 1 mole 2 15 60 rifampin 178 ; . The same amount of antibiotic minutes is bound by the core enzyme azo' ; , which is FIG. 3. Stability of the complex between rifampin also inhibited to the same degree as the holo and crude 0 ; or purified A ; RNA polymerase. In a enzyme 33, 178 ; . Thus, it is not o- that binds first step, the enzyme-'4C-rifampin complex was formed rifampin. Experiments with isolated subunits and isolated as previously described 176 ; . The stability have proved that rifampin binds to the i3 subunit of this complex was measured either by determining the rifampin with excess of added 193 ; . This result has been confirmed by experi- exchange ofthe labeled by adsorption an dissociated 14Ccold rifampin 178 ; or ments with RNA polymerase from rifamycin- rifampin to charcoal and measuring of remaining rifthe resistant cells see below ; . The formation of the amycin-enzyme complex Wehrli et al., in preparation ; . complex is very rapid: with 1.5 nmoles of enzyme per ml, a threefold excess of rifampin yields 95 % of the complex at 0 C less than 2 min Fig. 2 ; . cannot be explained yet, but it could be assumed The stability of the complex varies according to either that the f3 subunit that is responsible for the temperature and can be determined by the binding of rifamycin is somehow modified or measuring the exchange rate between unlabeled, that other factors that are lost during purification complexed rifampin and '4C-labeled, free rifampin contribute to the stability of the complex. Modifi 176, 178 ; . Furthermore, during enzyme purifica- cations of the enzyme by adenylation 24 ; or tion, the stability of the complex diminishes. phosphorylation 100 ; have recently been deWhereas a crude enzyme preparation forms a scribed. complex with rifampin that is practically stable Resistance Against the Action of Rifamycins for 1 hr at complex with a pure enzyme In an overnight culture of bacteria, cells reis decomposed to the extent of 90% under the sistant to rifamycins can be observed. The mutasame conditions Fig. 3 ; . The decrease of the complex stability due to enzyme purification tion rate for this resistance is 1.3 X 10-8 in the case of E. coli at a selection concentration of 100 , ug of antibiotic per ml, and the degree of resistance varies especially at low doses of drug. With -.S. high rifamycin concentrations, mutants showing * 90 o' complete resistance to the drug can be selected E 76 ; . find the reason for this resistance, RNA I 0 polymerase from totally resistance strains of E. 70. I coli and S. aureus have been isolated and tested 0. 7 I respect to their interaction with rifampin. The resistance proved to be due to a modified RNA E 50 I polymerase that was insensitive to the inhibitory l J action of rifampin 175 ; . Concentrations up to 0 301, 000 times higher than those inhibiting a sensitive E RNA polymerase showed no effect on the resistant enzymes, whereas actinomycin inhibited N 10. both sensitive and resistant enzymes to the same degree 175 ; . Inactivation of rifamycins by destruction or binding to factors in the mutant 5 10 0.5 extract could be excluded, since in a mixture of minutes sensitive and resistant enzyme a normal inhibiFIG. 2. Kinetics of rifampin-RNA polymerase com- tion of the sensitive enzyme was found 37 ; . These plex formation. Experimental conditions as previously experiments validate the conclusion that the described 176 ; . rifamycins interact specifically with RNA polyx and cholestyramine.
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LITERATURE CITED Austin, B., Rayment, J. 1985 ; . Epizootiology of Renibacterium salmoninarum, the causal agent of bacterial kidney disease in salmonid fish. J . Fish Dis. 8: 505-509 Daly, J. G . , Stevenson, R. M. \V. 1985 ; . Charcoal agar, a new growth medium for the fish disease bactenum Renibacterium salmon~narum. Appl. environ. Microbiol. 50: 868-87 1 Daly, J. G., Stevenson, R. M. W. 1987 ; . Hydrophobic and haemagglutinating properties of Renibacterium salmoninarum. J . gen. Microbiol. 133: 3575-3580 Evelyn, T. P. T. 1977 ; . An improved growth medium for the kidney disease bacterium and some notes on using the medium. Bulletin d e 1'Office International des Epizooties 87: 511-513 Evelyn, T. P. T., Ketcheson, J. E., Prosperi-Porta, L. 1981 ; .The clinical sign~ficanceof imn~unofluorescence-based diagnoses of the bacterial kidney disease carner Fish Pathol. 15: 293-300 Fryer, J. L., Sanders, J. E. 1981 ; . Bacterial ludney l s e salmonid fish. Ann. Rev. Microbiol. 35: 273-298 and chlorambucil.
Chase P, Walter F, James S. Whole bowel irrigation, hemodialysis, and continuous venovenous hemodiafiltration in the successful treatment of severe salicylate poisoning: Case report. Dialysis and Transplantation 2002; 31: 387 + . Chui PT. Anesthesia in a patient with undiagnosed salicylate poisoning presenting as intraabdominal sepsis. J Clin Anesth 1999; 11: 251-253. Cohen A, Thomas GB, Cohen EB. Serum concentration, safety and tolerance of oral doses of choline magnesium trisalicylate. Curr Ther Res Clin Exp 1978; 23: 358367. Cohen A. Comparison of two oral dosage forms of choline magnesium trisalicylate: bioavailability bioequivalence study. Curr Ther Res Clin Exp 1980; 27: 692698. Coldwell BB, Boyd EM. The acute rectal toxicity of acetylsalicylic acid. Can J Physiol Pharmacol 1966; 44: 909-918. Comstock EG, Boisaubin EV, Comstock BS, Faulkner TP. Assessment of the efficacy of activated charcoal following gastric lavage in acute drug emergencies. J Toxicol Clin Toxicol 1982; 19: 149165. Connell JR. Babies, dehydration, and aspirin. Rocky Mt Med J 1969; 66: 5761. Coombs FS, Warren HA, Higley CS. Toxicity of salicylates. J Lab Clin Med 1945; 30: 378379. Coppack SW, Higgens CS. Algorithm for modified alkaline diuresis in salicylate poisoning. Br Med J 1984; 289: 1452. Corby DG, Decker WJ, Anderson FP, Rowe DS. Therapy of acute salicylate ingestion. J Pediatr 1969; 75: 1083-1084. Cotton EK, Fahlberg VI. Hypoglycemia with salicylate poisoning. A report of two cases. J Dis Child 1964; 108: 171173. Craig JO, Ferguson IC, Syme J. Infants, toddlers, and aspirin. Br Med J 1966; 5490: 757761. Cumming G., Dukes DC, Widdowson G. Alkaline diuresis in treatment of aspirin poisoning. Br Med J 1964; 2: 10331036. Curtis RA, Barone J, Giacona N. Efficacy of ipecac and activated charcoal cathartic. Prevention of salicylate absorption in a simulated overdose. Arch Intern Med 1984; 144: 48 Czeizel AE, Rockenbauer M, Mosonyi A. Population based case control teratologic study of acetylsalicylic acid treatments during pregnancy. Pharmacoepidemiol Drug Saf 2000; 9: 193-205. Danel V, Henry JA, Glucksman E. Activated charcoal, emesis, and gastric lavage in aspirin overdose. Br Med J Clin Res Ed ; 1988; 296: 1507. Dargan PI, Wallace CI, Jones AL. An evidence based flowchart to guide the management of acute salicylate aspirin ; overdose. Emergency Medicine Journal 2002; 19: 206-209. Davies MG, Briffa DV, Greaves MW. Systemic toxicity from topically applied salicylic acid. Br Med J 1979; 1: 661. Davis DA, Kraus AL, Thompson GA, Olerich M, Odio MR. Percutaneous absorption of salicylic acid after repeated 14-day ; in vivo administration to normal, acnegenic or aged human skin. J Pharm Sci 1997; 86: 896-899. Davis PR, Burch RE. Pulmonary edema and salicylate intoxication. Ann Intern Med 1974; 80: 553554. Davison C, Zimmerman EF, Smith PK. On the metabolism and toxicity of methyl salicylate. J Pharmacol Clin Ther 1961; 132: 207211. Dellinger TM, Livingston HM. Aspirin burn of the oral cavity. Ann Pharmacother 1998; 32: 1107. Diamond EF, DeYoung VR. Acute poisoning with oil of wintergreen treated by exchange transfusion. AMA J Dis Child 1958; 95: 309310. Dickinson G, Yeats A, MacPhail I, Tierney M. Salicylate poisoning. Can Med Assoc J 1992; 147: 1426-1427. Done AK. Aspirin overdosage: incidence, diagnosis, and management. Pediatrics 1978; 62: 890897 and chondroitin.
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